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Figure 7: Adenosine-induced calcium influx in HEK-GHSR-1a

Figure 7: Adenosine-induced calcium influx in HEK-GHSR-1a cells. Calcium measurements were performed in cells suspended in nominally calcium-free medium

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hormone as an agonist of the ghrelin receptor GHS-R1a -

calcium mobilization in HEK 293 cells stably transfected with GHS-R1a an Metal ion-mediated agonism and agonist enhancement in melanocortin MC1 and

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Isolation and Confirmation of a Calcium Excretion

24 h) and WKY controls (2.11 ± 0.50 mg/24 h); the congenics preserve the calcium excretion phenotype of the GHS parent strain

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2GHS: CRYSTAL STRUCTURE OF A CALCIUM-BINDING PROTEIN, REGUCALCIN (AGR_C_1268) FROM AGROBACTERIUM TUMEFACIENS STR. C58 AT 1.55 A RESOLUTION 2GHS CRYS

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of the Growth Hormone Secretagogue Receptor (GHS-R1a)

but not with the partially edited 5-HT2C-VSV isoform, significantly reduced GHS-R1a agonist-mediated calcium influx, which was completely restored following

_MSDS___CAS【7790-75-2】_

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Remediated Sequence - 2GHS: CRYSTAL STRUCTURE OF A CALCIUM-

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to the growth hormone secretagogue receptor type-1a (GHS-

energy homeostasis through the GH secretagogue receptor type-1a (GHS-R1a). The results revealed that calcium rise of both ghrelin and adenosine appears